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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells SLC25A39 binds and modulates PRDX1 to suppress ROS-induced necroptosis in hepatocellular carcinoma
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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells SLC25A39 binds and modulates PRDX1 to suppress ROS-induced necroptosis in hepatocellular carcinoma
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l Cysteine Glutathione Mixed Disulfide, a Novel Bioavailable Sulfhydryl Modified Glutathione Precursor, Protects against Early Liver Injury Induced by Short Term Hypercholesterolemia Chemical Research in Toxicology SLC25A39 identified as a key regulator of hepatocellular carcinoma progression through the mitochondrial ROScytochrome ccaspase signaling axis Cellular & Molecular Biology Letters Springer Nature Link Mitochondrial GSH depletion sensitizes cells to BSO treatment a, Scheme Download Scientific Diagram Autoregulatory control of mitochondrial glutathione homeostasis Science SLC25A39 is necessary for mitochondrial glutathione import in mammalian cells Nature
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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells SLC25A39 binds and modulates PRDX1 to suppress ROS-induced necroptosis in hepatocellular carcinoma
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